 Receptor, Angiotensin, Type 1
"Receptor, Angiotensin, Type 1" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An angiotensin receptor subtype that is expressed at high levels in a variety of adult tissues including the CARDIOVASCULAR SYSTEM, the KIDNEY, the ENDOCRINE SYSTEM and the NERVOUS SYSTEM. Activation of the type 1 angiotensin receptor causes VASOCONSTRICTION and sodium retention.
| Descriptor ID |
D044140
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| MeSH Number(s) |
D12.776.543.750.100.047.625 D12.776.543.750.750.130.750
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| Concept/Terms |
Receptor, Angiotensin, Type 1- Receptor, Angiotensin, Type 1
- Receptor, Angiotensin II Type 1
- Angiotensin Type 1 Receptor
- Angiotensin AT1 Receptor
- AT1 Receptor, Angiotensin
- Receptor, Angiotensin AT1
- Angiotensin II Type 1 Receptor
Receptor, Angiotensin, Type 1b- Receptor, Angiotensin, Type 1b
- Receptor, Angiotensin II Type 1b
- Angiotensin Type 1b Receptor
- Angiotensin AT1b Receptor
- AT1b Receptor, Angiotensin
- Receptor, Angiotensin AT1b
- Angiotensin II Type 1b Receptor
Receptor, Angiotensin, Type 1a- Receptor, Angiotensin, Type 1a
- Receptor, Angiotensin II Type 1a
- Angiotensin Type 1a Receptor
- Angiotensin AT1a Receptor
- AT1a Receptor, Angiotensin
- Receptor, Angiotensin AT1a
- Angiotensin II Type 1a Receptor
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Below are MeSH descriptors whose meaning is more general than "Receptor, Angiotensin, Type 1".
- Chemicals and Drugs [D]
- Amino Acids, Peptides, and Proteins [D12]
- Proteins [D12.776]
- Membrane Proteins [D12.776.543]
- Receptors, Cell Surface [D12.776.543.750]
- Receptors, G-Protein-Coupled [D12.776.543.750.100]
- Receptors, Angiotensin [D12.776.543.750.100.047]
- Receptor, Angiotensin, Type 1 [D12.776.543.750.100.047.625]
- Receptors, Peptide [D12.776.543.750.750]
- Receptors, Angiotensin [D12.776.543.750.750.130]
- Receptor, Angiotensin, Type 1 [D12.776.543.750.750.130.750]
Below are MeSH descriptors whose meaning is more specific than "Receptor, Angiotensin, Type 1".
This graph shows the total number of publications written about "Receptor, Angiotensin, Type 1" by people in this website by year, and whether "Receptor, Angiotensin, Type 1" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1998 | 0 | 1 | 1 | | 2001 | 0 | 2 | 2 | | 2003 | 0 | 2 | 2 | | 2005 | 1 | 0 | 1 | | 2008 | 2 | 0 | 2 | | 2010 | 1 | 0 | 1 | | 2011 | 1 | 3 | 4 | | 2012 | 1 | 0 | 1 | | 2014 | 1 | 0 | 1 |
To return to the timeline, click here.
Below are the most recent publications written about "Receptor, Angiotensin, Type 1" by people in Profiles.
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Bhatt SR, Lokhandwala MF, Banday AA. Vascular oxidative stress upregulates angiotensin II type I receptors via mechanisms involving nuclear factor kappa B. Clin Exp Hypertens. 2014; 36(6):367-73.
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Chugh G, Lokhandwala MF, Asghar M. Altered functioning of both renal dopamine D1 and angiotensin II type 1 receptors causes hypertension in old rats. Hypertension. 2012 May; 59(5):1029-36.
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Javkhedkar AA, Lokhandwala MF, Banday AA. Defective nitric oxide production impairs angiotensin II-induced Na-K-ATPase regulation in spontaneously hypertensive rats. Am J Physiol Renal Physiol. 2012 Jan 01; 302(1):F47-51.
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Asghar M, Tayebati SK, Lokhandwala MF, Hussain T. Potential dopamine-1 receptor stimulation in hypertension management. Curr Hypertens Rep. 2011 Aug; 13(4):294-302.
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Banday AA, Lokhandwala MF. Angiotensin II-mediated biphasic regulation of proximal tubular Na+/H+ exchanger 3 is impaired during oxidative stress. Am J Physiol Renal Physiol. 2011 Aug; 301(2):F364-70.
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Banday AA, Lokhandwala MF. Oxidative stress causes renal angiotensin II type 1 receptor upregulation, Na+/H+ exchanger 3 overstimulation, and hypertension. Hypertension. 2011 Mar; 57(3):452-9.
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Chugh G, Lokhandwala MF, Asghar M. Oxidative stress alters renal D1 and AT1 receptor functions and increases blood pressure in old rats. Am J Physiol Renal Physiol. 2011 Jan; 300(1):F133-8.
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Banday AA, Lokhandwala MF. Loss of biphasic effect on Na/K-ATPase activity by angiotensin II involves defective angiotensin type 1 receptor-nitric oxide signaling. Hypertension. 2008 Dec; 52(6):1099-105.
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Banday AA, Lokhandwala MF. Oxidative stress-induced renal angiotensin AT1 receptor upregulation causes increased stimulation of sodium transporters and hypertension. Am J Physiol Renal Physiol. 2008 Sep; 295(3):F698-706.
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Banday AA, Siddiqui AH, Menezes MM, Hussain T. Insulin treatment enhances AT1 receptor function in OK cells. Am J Physiol Renal Physiol. 2005 Jun; 288(6):F1213-9.
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