 beta-Adrenergic Receptor Kinases
"beta-Adrenergic Receptor Kinases" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
G-protein-coupled receptor kinases that mediate agonist-dependent PHOSPHORYLATION and desensitization of BETA-ADRENERGIC RECEPTORS.
| Descriptor ID |
D051552
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| MeSH Number(s) |
D08.811.913.696.620.682.700.364.049 D12.644.360.293.249 D12.776.476.293.249
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| Concept/Terms |
beta-Adrenergic Receptor Kinases- beta-Adrenergic Receptor Kinases
- Receptor Kinases, beta-Adrenergic
- beta Adrenergic Receptor Kinases
- beta-Adrenergic Receptor Kinase
- Receptor Kinase, beta-Adrenergic
- beta Adrenergic Receptor Kinase
- beta-AR Kinase
- beta AR Kinase
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Below are MeSH descriptors whose meaning is more general than "beta-Adrenergic Receptor Kinases".
Below are MeSH descriptors whose meaning is more specific than "beta-Adrenergic Receptor Kinases".
This graph shows the total number of publications written about "beta-Adrenergic Receptor Kinases" by people in this website by year, and whether "beta-Adrenergic Receptor Kinases" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1995 | 0 | 1 | 1 | | 1996 | 0 | 1 | 1 | | 1999 | 0 | 1 | 1 | | 2004 | 0 | 1 | 1 | | 2005 | 1 | 1 | 2 | | 2006 | 4 | 3 | 7 | | 2007 | 4 | 1 | 5 |
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Below are the most recent publications written about "beta-Adrenergic Receptor Kinases" by people in Profiles.
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Banday AA, Fazili FR, Lokhandwala MF. Insulin causes renal dopamine D1 receptor desensitization via GRK2-mediated receptor phosphorylation involving phosphatidylinositol 3-kinase and protein kinase C. Am J Physiol Renal Physiol. 2007 Sep; 293(3):F877-84.
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Salim S, Hite B, Eikenburg DC. Activation of the CRF(1) receptor causes ERK1/2 mediated increase in GRK3 expression in CATH.a cells. FEBS Lett. 2007 Jul 10; 581(17):3204-10.
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Banday AA, Lokhandwala MF. Oxidative stress reduces renal dopamine D1 receptor-Gq/11alpha G protein-phospholipase C signaling involving G protein-coupled receptor kinase 2. Am J Physiol Renal Physiol. 2007 Jul; 293(1):F306-15.
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Banday AA, Fazili FR, Lokhandwala MF. Oxidative stress causes renal dopamine D1 receptor dysfunction and hypertension via mechanisms that involve nuclear factor-kappaB and protein kinase C. J Am Soc Nephrol. 2007 May; 18(5):1446-57.
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Salim S, Standifer KM, Eikenburg DC. Extracellular signal-regulated kinase 1/2-mediated transcriptional regulation of G-protein-coupled receptor kinase 3 expression in neuronal cells. J Pharmacol Exp Ther. 2007 Apr; 321(1):51-9.
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Salim S, Eikenburg DC. Role of 90-kDa heat shock protein (Hsp 90) and protein degradation in regulating neuronal levels of G protein-coupled receptor kinase 3. J Pharmacol Exp Ther. 2007 Mar; 320(3):1106-12.
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Matkovich SJ, Diwan A, Klanke JL, Hammer DJ, Marreez Y, Odley AM, Brunskill EW, Koch WJ, Schwartz RJ, Dorn GW. Cardiac-specific ablation of G-protein receptor kinase 2 redefines its roles in heart development and beta-adrenergic signaling. Circ Res. 2006 Oct 27; 99(9):996-1003.
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Moore RH, Millman EE, Godines V, Hanania NA, Tran TM, Peng H, Dickey BF, Knoll BJ, Clark RB. Salmeterol stimulation dissociates beta2-adrenergic receptor phosphorylation and internalization. Am J Respir Cell Mol Biol. 2007 Feb; 36(2):254-61.
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Fardoun RZ, Asghar M, Lokhandwala M. Role of oxidative stress in defective renal dopamine D1 receptor-G protein coupling and function in old Fischer 344 rats. Am J Physiol Renal Physiol. 2006 Nov; 291(5):F945-51.
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Desai AN, Salim S, Standifer KM, Eikenburg DC. Involvement of G protein-coupled receptor kinase (GRK) 3 and GRK2 in down-regulation of the alpha2B-adrenoceptor. J Pharmacol Exp Ther. 2006 Jun; 317(3):1027-35.
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